Do deficits with interhemispheric transfer of haptic information result from closed head injury?

We examined the interhemispheric transfer of haptic information in 21 closed head injury (CHI) survivors and 22 control subjects. The tactual recognition test was administered under two conditions: Simultaneous, in which subjects had access to target stimuli during the task, or Successive, in which subjects did not have access to test stimuli. Deficits in interhemispheric transfer of haptic information were apparent in the more memory-dependent condition. The results suggest that in CHI the callosal damage is sufficient to have transfer deficits when the combined demands of callosal transfer and memory are present.